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These infusion rates produce plasma levels similar to those seen during corticotropin stimulation.
2
For example, corticotropin-releasing factor regulates the homeostatic pattern of visceral functions during stress.
3
Concentrations of endorphins and corticotropin increased significantly also during the anaerobic exercise test.
4
A Sheehan event probably occurred 33 years before the patient presented with corticotropin deficiency.
5
Thirty-one patients underwent also a human corticotropin releasing hormone test.
6
As with corticotropin there were only minor changes in the coronary and iliac vascular beds.
7
A rate-sensitive fast-feedback inhibition of stress-induced corticotropin secretion by glucocorticoids is well documented in rats.
8
By contrast, Thy1-alphaSyn mice had an increased fecal response to novelty stress and corticotropin releasing factor injected intraperipherally.
9
Three of seven cases contained cells reactive for ACTH and corticotropin-like intermediate lobe peptide CLIP or their precursor, pro-opiomelanocortin.
10
This blunted neuronal response was also associated with an attenuated stimulation of neuroendocrine corticotropin-releasing factor transcription and plasma corticosterone release.
11
Objective: To describe the course of autoimmune hyperthyroid disease in a patient with corticotropin (ACTH) deficiency treated with glucocorticoids.
12
These findings suggest that disruption of NO synthesis does not play a role in the development of corticotropin hypertension in sheep.
13
Peak cortisol responses to corticotropin releasing factor and adrenocorticotropic hormone were correlated with each other and with 24-hour urinary-free cortisol excretion.
14
The activation of brain corticotropin-releasing factor (CRF) pathways has a key role in the behavioral and visceral responses to stress.
15
Objective: To investigate whether genetic variants of the corticotropin-releasing factor system or their interaction influence the risk of developing AUD in chronic disease populations.
16
The possibility that corticotropin (ACTH)-induced hypertension results from a direct central effect of the adrenocortical steroids released by ACTH was investigated in sheep.